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What Are Diabetes Nerve Damage Symptoms @ Complications Of Diabetes Nerve Damage Symptoms!

What are Diabetes Nerve Damage Symptoms @ Complications Of Diabetes Nerve Damage Symptoms! Click below to discover the secret to reverse your diabetes so you can enjoy your favorite food again.

What are Diabetes Nerve Damage Symptoms @ Complications Of Diabetes Nerve Damage Symptoms! When you leave your doctor’s office, do you ever wonder what he’s not telling you? Every appointment, he tells you to eat less sugar and go on more walks, but aren’t there other ways to get your diabetes under control? Here are 3 tricks to manage your diabetes that your doctor won’t tell you: 1- Eat More Fat You read that right. Eat more fat. That’s because fat helps your body absorb insulin. That means the more fat you eat, the easier it’ll be to manage your blood sugar. But here’s the kicker: It’s got to be the right type of fat.

You’re looking for Unsaturated Omega-3 Fat. Here are some great sources: Fish Eggs (Any eggs labeled “enriched” have plenty of omega-3) Grass-fed beef (There’s lots of omega-3 in the grass) Do Some Pushups… Or any kind of strength exercises. All the cardio your doctor tells you to do will increase your insulin absorption a little, but to really keep your body regulated you’ve got to get your entire body moving. The best way to do that is an exercise that focuses on strength. You want to avoid straining yourself, but make a habit of doing a few pushups every day, throw in somebody squats, and soon you’ll be taking tighter control of your blood sugar. Not to mention it’ll get rid of stress, and give you plenty of energy. Relax Laying back and keeping cool are vital to regulating your blood sugar. Stress causes physical distress on the body which affects blood glucose levels. Not to mention, when you’re stressed out it’s easy to overeat, which obviously wreaks havoc on your blood sugar. For easy relaxation, try out simple meditation or breathing exercises.


Mild Treatment For Diabetes

Acupressure points to cure diabetes acupressure is one of the natural ways to cure your diabetes and has been sought-after over allopathy due to its healing properties by using body’s own energy

diabetes is of two basic types type one where the pancreas in one’s body does not produce enough insulin to digest sugars and type 2 where the cells become immune to the insulin and thus do not digest sugar that’s outside the blood either ways blood sugar levels are affected giving results to a lot of adverse reactions in the body but the good news is diabetes can be controlled to a large extent if not totally cured outer pressure is an alternative treatment to cure various diseases

there are hundreds of acupressure points in our body by putting physical pressure on the right acupressure points one can not only relieve the stress and tension but can also get rid of various diseases including the blood sugar

acupressure is helpful for both the pre-diabetics and the Tibetans our Capricia along with healthy diet and her’ll blood sugar management medicines can help following

acupressure points cannot only help relieve the pain for the diabetics but can also help to prevent blood sugar wrists handy this occupation point basically lies near the wrist the side of the wrist where the little finger of your hand lies the right side of your wrist on your left hand and the left side of your wrist on your right hand is where this point lies as shown in video massage and apply occupation on this point every day before your meals for five minutes which will control your diabetes automatically feet there are specific points on the feet that control problems like hypertension while treat diabetes as well as insomnia

this occupation quite lies on the front side of your foot a little before your largest two on the second toe apply pressure under the big toe with the help of your fingers since it is hard for you to do this alone ask a friend to help the pressure points under the big toe will help in giving a long-term relief from blood sugar pressing this point for 2 to 3 minutes every day will help relieve symptoms of blood sugar repeat on both feet the occupation points will help you manage diabetes symptoms by managing the metabolic process and maintaining the insulin level

mild pressure exertion on occupation points along with proper diet and exercise improves metabolism and insulin levels bitter gout how to make the juice of bitter gout a common vegetable used in day to day Indian main course put the juice in a white bucket then immerse your feet in the juice and keep paddling very softly on the juice for about fifteen to thirty minutes every day for minimum twenty to twenty-five days the duration to soak the feet in the juice is till when you feel the bitterness spit all the way to your tongue s you read it right the bitterness feeling will rise from your feet

all the way to your throat giving you a bitter feeling as if you just had raw bitter gout this is a proven and tested method to reduce blood sugar level up to 100 to 150 points this method is 100% natural cure for type 2 diabetes patients were 100 people have reported they got cured 99% by applying to this method however to get maximum benefit from this method

it is also suggested to drink bitter gourd juice half glass every day early in the morning immediately after you are awake before you even brush you teeth a routine physical activity like walking or slow jogging for at least 20 to 30 minutes having enough sleep minimum seven arts sleeping early at least at 10 p.m. and waking up early avoid sleeping late if you have any work which you need to finish better wake up early undo it this way your body will thank you and you will help to focus more on your tasks before applying to this method test your blood sugar keep a record of it then test every three days and see the difference

this method will also show good results for people with type 1 diabetes the blood sugar level decreased significantly after applying to this method please do apply the above described a method in your routine warning this information is solely for informational purposes it is not intended to provide medical advice and should not be treated as a substitute for the medical advice of your own doctor.


Gastroparesis Solution to BG spikes for a Type 1 Diabetic

Occasionally, in the middle of my sleep cycle, I was having an abrupt increase in blood sugar I have no doubt this is a result of my gastroparesis diabeticorum (delayed stomach emptying) due to a neuropathy caused by poor glycemic control of my type 1 diabetes

I tried several methods in Dr. Bernstein’s Diabetes Solution regarding this issue including: chewing gum stomach exercises Betaine HCL with pepsin digestive enzyme capsules and prescription Zofran. the only thing that helped was the Zofran. but at a side effect that made this medication unworthy of a long-term solution I do not recommend it. Another solution would be liquid meals, but I look forward to my meals And I didn’t want to explore this venue unless absolutely necessary. Let me explain my day and eating schedule. I work 3rd shift. I awake at p.m. Eat dinner between and p.m.

Eat lunch about AM Then eat breakfast at to AM. I go to sleep around PM. The gastroparesis spike, when it happens, occurs between 4 to 6 p.m. my solution was figured out and tested after I fasted for blood tests and explained to my doctor that what this was and how I tried to treat it My dinner, the last meal I ate before my fast, the night before my bloodwork did not empty out of my stomach for about nine hours. If I eat a big lunch six hours after dinner, My guess is that my stomach stops and slows digestion. Then again eating breakfast about five hours later can slow and stop my digestion once more.

After I’m asleep, between 4 & 6 PM, my stomach empties causing a surge in blood sugar I usually awake to a high Dexcom CGM warning, take a correction dose of insulin, and go back to sleep I skip lunch on the weekends and do not have these spikes in blood sugar during my sleep Cycle. I performed an experiment. Instead of my regular lunch of a ‘Whip It’ bread sandwich and 2 ounces of cheese, I only ate the 2 ounces of cheese for lunch It’s an easily digested and small protein meal.

It worked! Ever since this change, I haven’t had a blood sugar spike during my sleep cycle. Normal glycemic control for a type one, insulin-dependent diabetic is a tricky business, but none-the-less achievable Just to recap what was happening before my dinner has emptyed from my stomach, I was eating a big lunch slowing or stopping digestion I was eating breakfast before the previous meals had emptyed my stomach slowing digestion once more. While in the middle of my sleep cycle, my stomach was empty causing a huge blood sugar spike! I fixed this by sizeably reducing and simplifying my lunch.

If you have this problem, take note of when your stomach empties and adjust. It certainly worked for me!.


Micro And Macrovascular Complications Of Diabetes

Now I want to talk about the complications caused by diabetes all of the complications of diabetes may be prevented by keeping blood sugars within an acceptable range and by keeping scheduled appointments with your healthcare specialists the acceptable blood sugar range is seventy to a 130 milligrams per deciliter before a meal and less than a hundred and eighty milligrams :00:38.150,0:00:42.190 per deciliter two hours after a meal

There are two overall types of diabetes complications microvascular and macrovascular.

Microvascular complications affect the small arteries the capillaries which are the smallest blood vessels that connect arteries and veins and the nerves such as those found in the eyes, kidney, feet skin and stomach damage to the retina

A part of the eye is known as retinopathy symptoms of retinopathy include problems when reading blurred vision seeing halos around lights seeing dark spots or light flashes damage from retinopathy can be prevented by having annual dilated eye exam done by an eye specialist such as an opthalmologist and having identify problems treated early retinopathy left untreated can lead to blindness the risk of developing both cataracts and glaucoma is also a higher for people with diabetes damaged to the kidney is known as nephropathy with nephropathy

the kidney cannot adequately eliminate waste in the urine this leads to retention of fluid in the body contributing to hide blood pressure dialysis may be necessary to filter the excess fluid and waste because of kidney insufficiency or failure diabetes can also cause infections of the feet and skin this is because wounds heal slower in some cases wounds on the feet if left untreated can lead to foot ulcers and in some cases require amputation damage to the nerves is known as neuropathy with neuropathy you may field tingling numbness or pain in the lower extremities feet or hands neuropathy may affect other parts of the body such as the digestive tract or urinary tract this can cause delayed gastric or stomach emptying or incomplete bladder emptying which can cause bladder or urinary tract infections

the second type of diabetes complication is called macro vascular this is damaged to the large arteries such as the carotid arteries and arota as well as internal organs in the brain these macro vascular effects can include damage to the heart into the circulatory system leading to heart disease or stroke symptoms of macro vascular complications include dizziness difficulty speaking weakness in the legs and arms chest pain or pressure irregular heartbeat and swollen ankles diabetes in reality is probably the most single devastating disease because of retinopathy, because of nephropathy, because of neuropathy which means it involves the kidney the eye, it involves all the nerves in your body and when you talk about neuropathy we think about the lower extremities, but every single nerve in the body could be involved in diabetes if you want to learn more about diabetes checkout other videos including treatment options


Gestational Diabetes Symptoms While Pregnant

Diabetes, it’s certainly not a problem that’s unique to pregnancy, but it’s something we always have to discuss when we’re talking about pregnancy because it can really complicate that picture quite a bit. So firstly, diabetes and pregnancy split up into two different categories. There’s the category of women who had diabetes before becoming pregnant, and that’s called pre-gestational diabetes. So that category is referred to as pre-gestational diabetes. Pre for before, gestational for pregnancy, so before pregnancy diabetes.

And then there are the women who become diabetic during their pregnancy, which is called gestational diabetes. So that’s the second category, gestational diabetes. And 90% of cases, so that’s nine of every 10 cases of diabetes in pregnancy falls into this category of gestational diabetes. And then the other 10% are pre-gestational diabetics. So there’s something about pregnancy that makes women more susceptible to developing diabetes and we’ll discuss exactly what that is in just a bit. But firstly, why do we even split up diabetes into these two different groups? Well, if a woman has pre-gestational diabetes, that means that her blood sugars may have been poorly controlled at the time that the baby was conceived.

Or even during the first eight weeks of the pregnancy, during a period called organogenesis. So the first eight weeks of pregnancy is a period called organogenesis, when the fetus’ organs are made. And those high levels of glucose during that really pivotal time can lead to a miscarriage or it can lead to significant anomalies within the fetus. However, with gestational diabetes, that problem with glucose control develops during the pregnancy, in some ways because of the pregnancy.

And usually the glucose control isn’t impaired until the second trimester, so after the point of conception and after the point of organogenesis. So miscarriage and fetal anomalies don’t tend to be a problem with gestational diabetes. But that’s not to say that gestational diabetes doesn’t harm the fetus. Rather, diabetes as a whole, so regardless of the category, can cause preterm labor. It can cause problems with the growth of the fetus. It can even lead to stillbirth. And one of the complications that we tend to think about a lot, that tends to be talked about a lot, is fetal macrosomia. So fetal macrosomia. So let me explain that a little bit. Alright, so if mom has diabetes, the basic gist of it all is that her glucose levels tend to run on the high side.

And glucose can cross the placenta, into the bloodstream of the fetus. That’s like one of the main purposes of the placenta, to allow glucose to enter the baby’s bloodstream as an energy supply. So then when mom has high glucose levels in her blood, then the fetus has high glucose levels in its blood. And that drives the release of insulin in the fetus, because that’s the body’s primary response to glucose, to release insulin. Insulin is kind of the key that allows cells to open up their doors and take up glucose and use it. And so that insulin that’s released in the fetus allows glucose to be taken up. And insulin does a few other things. It also stimulates fat storage in the body, right? And it also binds to receptors on different organs, such as the heart and the liver, and it causes them to grow. It causes the organs to actually grow in size. And so the end result of it all, the end result of the high glucose levels in the mom, leading to high glucose levels in the baby, leading to high insulin levels in the baby, is that the baby grows to a larger size than normal, which is called fetal macrosomia.

Macro for large and soma for body. So larger body. Now another thing that I want to mention is that in pre-gestational diabetes, so again, diabetes before the point of pregnancy, the impaired glucose control is more long-standing. So these women are more likely to have diabetic complications such as kidney damage or vascular problems, so blood vessel related problems, and damage to the retina. And pregnancy can aggravate these complications, so it can it make worse. So it’s really important to monitor these conditions throughout the pregnancy. Now, I want to stop dancing around the issue of why women can become diabetic during pregnancy. A lot of it has to do with the hormones that are released during pregnancy. So hormones such as HPL, that’s not one that many people have heard of, right? It stand for human placental, human placental lactogen.

Alright, so that’s HPL. Another hormone is cortisol. So the body’s main stress hormone. Another one that you may have heard of before is growth hormone, that’s released in a large quantity during pregnancy. And then finally, progesterone. Progesterone, which is exceptionally important for the maintenance of a healthy pregnancy. So these hormones are released during pregnancy and they have lots of important roles. And among their many, many roles, these hormones increase mom’s production of glucose during pregnancy to make sure that the fetus has enough of the glucose, enough of its primary fuel source. And that leads to high glucose levels within the mom’s blood. And you might be thinking, “Well, that’s no problem, “because the glucose will cause insulin to be released “and that insulin will cause mom’s cells “to take up the glucose and problem solved. “You don’t have high glucose levels in the blood any more.” Well, unfortunately, it doesn’t really work that way, because these same hormones make the mom’s body resistant to insulin. So that the cells don’t respond to insulin and don’t take up as much glucose from the blood.

And this is done for a purpose. It’s actually done so that you can reduce mom’s utilization of the glucose so that more of the glucose is available for the fetus. And that’s why you can end up with high blood glucose levels and diabetes in pregnancy. Or if you had diabetes before you became pregnant, it can become worse during pregnancy. So, given that diabetes can cause all of these complications for mom and baby during pregnancy, it goes without saying that we do our absolute best to screen for it during pregnancy.

So for women who have a normal risk of having diabetes, we do a routine screen. So for all women, all average women, we do a routine screen around 26 to 28 weeks into the pregnancy. And that screening test is usually in the form of a glucose tolerance test. So the screening test is often called the glucose tolerance test, where the woman is given a very specific amount of glucose and her blood glucose levels are measured at one, two, or three hours after consuming that very specific amount of glucose. And if her blood glucose levels are above the normal range, then she’s found to be diabetic.

And if a woman is diagnosed with diabetes during her pregnancy, we do our best to control it with diet. And if that doesn’t work, then insulin is kind of our second line of treatment. And it’s also important to know that gestational diabetes, so again, diabetes that occurs during pregnancy, kind of as a process of pregnancy, increases the risk of a woman having overt diabetes after the pregnancy is over. So it’s really important to follow up with all these women after they deliver. Kind of the set point, six weeks after they deliver to test them for diabetes. Okay, so that is gestational diabetes in a nut shell.


Leaking Kidneys Diabetes

– One of the most serious chronic complications of diabetes mellitus is a condition known as diabetic nephropathy. Which, if you break down the term into nephro and pathy literally means kidney disease that occurs secondary to diabetes. And it’s actually pretty common as it eventually affects about 20% to 40% of all individuals with diabetes, including both type I and type II. In this tutorial, let’s talk about the mechanism underlying the cause of diabetic nephropathy and how individuals with diabetes develop the condition. So diabetic nephropathy is a chronic complication of diabetes mellitus. Meaning, it usually has a slow progression over decades after the initial diagnosis of diabetes. And to give you an overview of what happens, an insulin deficiency due to the diabetes results in hyperglycemia, which then causes hypertension and kidney dysfunction. This kidney function is actually then further worsened by the hypertension. And ultimately, all of this results in kidney failure, which can have very severe and potentially even life threatening complications, such as anemia, electrolyte imbalances, such as metabolic acidosis, and heart arrhythmias.

Now, before we dive into the mechanism of diabetic nephropathy, let’s briefly review the structure of the glomerulus in the kidney, by bringing in a diagram here. So, the glomerulus is the portion of the kidney where blood is initially filtered. So blood enters the glomerulus over here, through this afferent arterial, and then leaves the glomerulus through the efferent arterial. And you can remember this, that it leaves through the efferent arterial for E for exit, or efferent. And while the blood is within the glomerulus, there’s this advanced filtration system, which we’ll talk about more in a minute. And the filtered fluid that exits the blood is known as a filtrate and it collects in Bowman’s space before it enters into the tubules of the nephron where further reabsorption and secretion occurs before it exits the kidney into the ureters as urine. Now, one last structure to point out in this diagram is this vessel coming off the efferent tubule, here.

Now, this vasculature actually wraps around the tubules of the nephron, and contributes to the reabsorption and secretion of solutes. Now, to add to this diagram, let’s imagine we took a cross section of this glomerulus, and looked at it on its end. And it would look a little bit something like this. Now, we can use this diagram here to better depict some of the important structures within the glomerulus. So here you can see the capillary vessels, and each of them I’ve drawn in here a little red blood cell to help remind you that it’s a blood cell.

And as you can see, these vessels are surrounded by a few additional structures that we couldn’t really appreciate in that first diagram. So these are the structures that contribute to the three layered filtration system of the glomerulus. The first layer is that of the vascular endothelium. So the endothelial cover, the inside of the blood vessel, so the capillary wall, there. And then the second layer is the glomerular basement membrane, or GBM for short, which is a specialized basement membrane that surrounds the vascular endothelium. And then the last filtration layer is the visceral epithelium, which is also known as the podocytes. Now, in between all these capillaries here is the mesangium, which is comprised of cells known conveniently as mesangial cells. And they produce a collagen network that structurally supports all of these capillaries and it’s across this space that filtration occurs within the glomerulus of the kidney. So how exactly does diabetes, a problem with insulin deficiency, result in kidney damage? Well, the answer includes multiple compounding factors.

Now, the first component is an increased pressure state within the nephron. And this is due to two mechanisms. And the first is hypertension, which is a common comorbidity associated with diabetes mellitus. So hypertension or high blood pressure results in an increased pressure throughout the entire arterial vascular system. And this includes the afferent arterial of the glomerulus. So, to think about how this increases the pressure within the glomerulus, let’s think about a simple garden hose. So, in the middle of the garden hose, there’s a hole. And as water flows through the hose, a small amount of water will leak out through this hole. But if we open up the spigot all the way this is going to increase the pressure of the water traveling through the hose, and intuitively, this change is going to result in more water leaking from the hole here in the center and that’s because there’s increased pressure forcing it out of the hole.

Now this is similar to what occurs in the glomerulus. The hypertension increases the pressure, just like turning on that spigot, which in return increases the filtration rate of the glomerulus, which can be thought of as that leakiness from the hole in the garden hose. Now, the other mechanism contributing to this high pressure state, is something known as vasoconstriction of the efferent arterial. Which is just a fancy way of saying that this blood vessel constricts or gets smaller in diameter. So, to understand why this occurs, we need to briefly review the renin-angiotensin-aldosterone system, or RAAS, for short.

So renin is a hormone that’s secreted by the kidneys in response to decreased renal profusion, or low blood flow to the kidney. This is a sign of low fluid volume throughout the body. So in the response to a low fluid volume, renin has a cascade of effects in order to maintain blood pressure as well as volume status. And one of these effects is constriction of the efferent arterial, which then maintains this pressure within the glomerulus in the presence of a decreased renal profusion. So once again, let’s go back to this garden hose to understand this a little bit better. Now, instead of turning up the spigot, as we did before, what do you think would happen if you were to kink the hose on the other side of the hole? Once again, intuitively, this is going to increase the pressure behind the kink and subsequently will increase the rate at which water leaks out the hole. So once again, this is similar to what occurs in the glomerulus in response to activation of this renin-angiotensin-aldosterone system. There’s a constriction of the efferent arterial to build up pressure within the glomerulus to maintain the necessary filtration and therefore, it will increase the filtration rate even further.

But why exactly is this happening? If I just said that individuals with diabetes often have increased renal profusion due to the hypertension, then why is a low pressure system such as the renin-angiotensin-aldosterone system activated? And this is a good question, and the answer is not exactly intuitive. For some reason, the underlying physiology of diabetes, specifically the hyperglycemia, results in a direct intrarenal or within the kidney activation of this renin-angiotensin-aldosterone system. And subsequently, efferent vasial constriction independent of the volume status of the individual and therefore increases the glomerular filtration rate. So how does this increased pressure relate to diabetic nephropathy? Well, as the pressure within the glomerulus increases, this results in a process known as mesangial expansion.

The increased pressure results in trauma and damage to the mesangium of the glomerulus. And in response to this damage, the mesangial cells respond by secreting cytokines that produce inflammation, as well as oxygen free radicals that result in endothelial dysfunction, and all of this kind of combines into hypertrophy and matrix accumulation within the mesangium, which is known as mesangial expansion. And as you can see over here on the right, as the mesangium expands, the spaces, or what are known as the fenestrations between the podocyte foot processes expand.

Now, this has two effects. First, it decreases the surface area available within the glomerulus for filtration, and second, the dilation of the fenestrations causes the filtration system to be leaky, and larger molecules such as proteins are filtered out of the blood in the kidney. Then, the last factor contributing to diabetic nephropathy is a combination of the previously mentioned factors. And this is ischemia. As I mentioned earlier, the blood vessels supplying the tubules of the nephron come off of the efferent arterial, and vasoconstriction of this arterial from the intrarenal activation of the renin-angiotensin-aldosterone system decreases this blood flow. And in addition, the cytokines and free radials produced from the barotrauma to the mesangium further damage the nephron vasculature. And over time these processes result in ischemia, or cell death, and atrophy of the vasculature that supports the glomerulus, as well as the tubules.

So this will decrease the kidney’s ability to filter blood, and is ultimately what will lead to kidney failure in diabetic nephropathy. So as you can see, there are many different mechanisms that are going to contribute to the progression of kidney failure in individuals with diabetes mellitus. However, it’s important to note that they are all directly associated with the underlying hyperglycemia, and therefore the progression towards kidney failure can be slow or potentially even prevented, if the underlying diabetes is well controlled.


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